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Rescue of myocardial energetic dysfunction in diabetes through the correction of mitochondrial hyperacetylation by honokiol

Abstract:

Cardiac energetic dysfunction has been reported in patients with type 2 diabetes (T2D) and is an independent predictor of mortality. Identification of the mechanisms driving mitochondrial dysfunction, and therapeutic strategies to rescue these modifications, will improve myocardial energetics in T2D. We demonstrate using 31P-magnetic resonance spectroscopy (31P-MRS) that decreased cardiac ATP and phosphocreatine (PCr) concentrations occurred before contractile dysfunction or a reduction in PC...

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Publication status:
Published
Peer review status:
Peer reviewed

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Publisher copy:
10.1172/jci.insight.140326

Authors


More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
ORCID:
0000-0001-5176-1764
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Physiology Anatomy & Genetics
Role:
Author
More by this author
Institution:
University of Oxford
Division:
MSD
Department:
Pharmacology
Role:
Author
ORCID:
0000-0001-9136-7518
More from this funder
Grant:
FS/10/002/28078
FS/14/17/30634
FS/19/18/34252
Publisher:
Journal of Clinical Investigation Publisher's website
Journal:
JCI insight Journal website
Volume:
5
Issue:
17
Article number:
e140326
Publication date:
2020-09-03
Acceptance date:
2020-07-16
DOI:
EISSN:
2379-3708
ISSN:
2379-3708
Pmid:
32879143
Language:
English
Keywords:
Pubs id:
1130742
Local pid:
pubs:1130742
Deposit date:
2020-09-07

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